18 May 1998 Retinal damage mechanisms from ultrashort laser exposure
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Proceedings Volume 3255, Applications of Ultrashort-Pulse Lasers in Medicine and Biology; (1998) https://doi.org/10.1117/12.308221
Event: BiOS '98 International Biomedical Optics Symposium, 1998, San Jose, CA, United States
Abstract
Extensive research of ultrashort ocular damage mechanisms has shown that less energy is required for retinal damage for pulses shorter than one nanosecond. Laser minimum visible lesion thresholds for retinal damage from ultrashort (i.e. < 1 ns) laser pulses occur at lower energies than in the nanosecond to microsecond laser pulse regime. WE review the progress made in determining the trends in retinal damage from laser pulses of one nanosecond to one hundred femtoseconds in the visible and near-infrared wavelength regimes. We discuss the most likely damage mechanism(s) operative in this pulse width regime and discuss implications on laser safety standards.
© (1998) COPYRIGHT Society of Photo-Optical Instrumentation Engineers (SPIE). Downloading of the abstract is permitted for personal use only.
Benjamin A. Rockwell, Dale J. Payne, Richard C. Hopkins, Daniel X. Hammer, Paul K. Kennedy, Rodney E. Amnotte, Brent Eilert, Jeffrey J. Druessel, Cynthia A. Toth, William P. Roach, Shana L. Phillips, David J. Stolarski, Gary D. Noojin, Robert J. Thomas, Clarence P. Cain, "Retinal damage mechanisms from ultrashort laser exposure", Proc. SPIE 3255, Applications of Ultrashort-Pulse Lasers in Medicine and Biology, (18 May 1998); doi: 10.1117/12.308221; https://doi.org/10.1117/12.308221
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