14 June 1999 Retinal damage mechanisms and safety for ultrashort laser exposure
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Proceedings Volume 3601, Laser-Tissue Interaction X: Photochemical, Photothermal, and Photomechanical; (1999) https://doi.org/10.1117/12.349987
Event: BiOS '99 International Biomedical Optics Symposium, 1999, San Jose, CA, United States
Abstract
For the past several years the US Air Force has led a research effort to investigate the thresholds and mechanisms for retinal damage from ultrashort laser pulses [i.e. nanosecond (10-9 sec) to femtosecond (10-15 sec) pulse widths]. The goal was to expand the biological database into the ultrashort pulse regime and thus to allow establishment of maximum permissible exposure limits for these lasers. We review the progress made in determining trends in retial damage by ultrashort laser pulses in the visible and near infrared, including variations in spot size and number of pulses. We also discuss the most likely damage mechanisms operative in this pulse width regime and discuss relevance to laser safety.
© (1999) COPYRIGHT Society of Photo-Optical Instrumentation Engineers (SPIE). Downloading of the abstract is permitted for personal use only.
Benjamin A. Rockwell, Benjamin A. Rockwell, Cynthia A. Toth, Cynthia A. Toth, William P. Roach, William P. Roach, Dale J. Payne, Dale J. Payne, Richard A. Hopkins, Richard A. Hopkins, Paul K. Kennedy, Paul K. Kennedy, David J. Stolarski, David J. Stolarski, Gary D. Noojin, Gary D. Noojin, Robert J. Thomas, Robert J. Thomas, Clarence P. Cain, Clarence P. Cain, } "Retinal damage mechanisms and safety for ultrashort laser exposure", Proc. SPIE 3601, Laser-Tissue Interaction X: Photochemical, Photothermal, and Photomechanical, (14 June 1999); doi: 10.1117/12.349987; https://doi.org/10.1117/12.349987
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